Kisunla (Donanemab-azbt) Comparison to Other Alzheimer Treatments

Donanemab-azbt (Kisunla) demonstrates distinct advantages and trade-offs compared to other Alzheimer's treatments, particularly monoclonal antibody therapies like lecanemab and aducanumab. Here's a structured comparison:

Mechanism of Action

  • Donanemab: Targets mature, aggregated amyloid-beta plaques (N-terminal pyroglutamate Aβ)[2][5].

  • Lecanemab: Binds soluble Aβ protofibrils earlier in plaque formation[2][6].

  • Aducanumab: Recognizes both fibrillar and soluble Aβ aggregates[4].

Cognitive & Functional Efficacy

Treatment

Cognitive Decline Reduction
(vs placebo)

Key Trial Findings

Donanemab

35% (low/medium tau group)[1][5]

39% lower disease progression risk; 47% halted progression at 1 year[5]

Lecanemab

27% (ADAS-Cog)[4]

Slower decline in daily living scores vs donanemab[3]

Aducanumab

22% (ADAS-Cog)[4]

Less effective than donanemab/lecanemab in network meta-analyses[4][6]

Donanemab showed 3× greater effect size than lecanemab/aducanumab on the Clinical Dementia Rating scale (CDR-SB) in head-to-head analyses[6].

Amyloid Clearance

  • Speed: Donanemab clears plaques faster (84% reduction at 18 months vs lecanemab’s 59% at 18 months)[5][6].

  • Dosing Protocol:

    • Donanemab: Monthly infusions with potential treatment cessation upon plaque clearance[5]

    • Lecanemab: Biweekly indefinite infusions[2]

Safety Profile

Adverse Event

Donanemab

Lecanemab

Aducanumab

ARIA-Edema (ARIA-E)

24%[1]

12.6%[3]

35%[4]

Microhemorrhages

25%[1]

17%[3]

19%[4]

Treatment-related deaths

0.3%[1]

0.7%[3]

0.4%[4]

Donanemab has higher ARIA risk than lecanemab but lower mortality risk[1][3]. All three antibodies show worse tolerability than placebo[4].

Practical Considerations

  1. Patient Selection:

    1. Donanemab works best in low/medium tau populations (no significant effect in high tau)[5]

    2. Requires APOE ε4 testing due to ARIA risk stratification[1]

  2. Monitoring Burden:

    1. Donanemab: Frequent MRIs (5 scans in first 7 months)[1]

    2. Lecanemab: Less intensive monitoring but lifelong dosing[2]

  3. Cost/Workflow:

    1. Donanemab’s potential finite treatment duration (6-18 months) may reduce long-term costs[5]

    2. Both require specialized infusion centers[2][5]

Emerging Comparisons

  • Lithium: Outperformed donanemab on MMSE in meta-analyses but lacks large-scale AD validation[4]

  • Combination Therapies: Research ongoing with tau-targeting drugs to address donanemab’s limited efficacy in high-tau patients[5]

Donanemab offers a time-limited treatment option with strong cognitive benefits for early AD patients, but requires careful risk-benefit analysis due to monitoring demands and ARIA risks. Lecanemab may be preferable for patients needing lower ARIA risk tolerance, while aducanumab lags in efficacy comparisons[3][4][6].


Citations:

[1] https://www.nia.nih.gov/news/nia-statement-donanemab-results-more-alzheimers-research-progress

[2] https://mylocalinfusion.com/donanemab-vs-lecanemab/

[3] https://dig.pharmacy.uic.edu/faqs/2024-2/sep-2024-faqs/what-evidence-supports-the-efficacy-and-safety-of-donanemab-for-alzheimer-disease/

[4] https://pubmed.ncbi.nlm.nih.gov/38253184/

[5] https://www.alzheimersresearchuk.org/news/new-alzheimers-drug-donanemab-what-is-it-and-how-does-it-work/

[6] https://www.medrxiv.org/content/10.1101/2024.03.31.24305134v1.full-text

[7] https://pmc.ncbi.nlm.nih.gov/articles/PMC11097689/

[8] https://www.drugs.com/medical-answers/how-decide-between-leqembi-kisunla-3578170/

[9] https://pmc.ncbi.nlm.nih.gov/articles/PMC11098549/

[10] https://www.explorationpub.com/Journals/en/Article/100648

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